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n-methyl-d-aspartate
1 headnote for n-methyl-d-aspartate
61 documents for n-methyl-d-aspartate
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Dr. [Gultekin] is the third Fulbright Scholar the Health Science Center has hosted in the past two years. Recipients of the prestigious Fulbright award are selected on the basis of academic or professional achievement and because they've demonstrated extraordinary leadership potential in their fields. "I've had the opportunity to work with several foreign researchers in my lab," Dr. [Russel Reiter, Ph.D.] said. "It is a pleasure for me to host Dr. Gultekin, as well. He is a scholarly individual with strong dedication and determination." Dr. Gultekin is examining the effects melatonin has on NMDA (N-methyl-D-aspartate) receptors in the brain.
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Adolescence is a period of rapid growth and physical change; a central question is whether consuming alcohol during this stage can disrupt development in ways that have long-term consequences. In general, the existing evidence suggests that adolescents rarely exhibit the more severe chronic disorders associated with alcohol dependence such as liver cirrhosis, hepatitis, gastritis, and pancreatitis. Adolescents who drink heavily, however, may experience some adverse effects on the liver, bone, growth, and endocrine development. Evidence also is mounting, at least in animal models, that early alcohol use may have detrimental effects on the developing brain, perhaps leading to problems with cognition later in life. This article summarizes the physiological effects of alcohol on adolescents...
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To: MEDICAL EDITORS Contact: Nicolas Bombourg of Reportlinker, US, +1-805-652-2626, Intl., +1-805-652-2626, nbo@reportlinker.com
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By fusing a mutant form of the rapamycin-binding FKBP12 to various proteins, the authors show that such chimeric proteins are subject to rapid degradation unless protected by binding a cell-permeable ligand, termed ShIdI. N. Manjunath Swamy from Harvard University Medical School and colleagues synthesized a peptide derived from rabies virus glycoprotein (RVG) and showed that the 29-amino-acid peptide bound specifically to acetylcholine receptors expressed in the brain.1 When they gave the RVG peptide (complexed with an antiviral) intravenously to mice, they silenced specific genes in the brain that were involved in an otherwise fatal form of viral encephalitis.
